R+ Alpha Lipoic

R+ Alpha Lipoic Acid protects and optimizes in particular the functioning of the mitochondria (area located inside the cell and a real energy cell, responsible for making fuel and optimizing its use by each of our cells from food).
Oxidation of the mitochondria plays a key role in wear and tear, slowing down and aging of the brain and body.

R+ Alpha Lipoic Acid protects and optimizes in particular the functioning of the mitochondria (area located inside the cell and a real energy cell, responsible for making fuel and optimizing its use by each of our cells from food).
Oxidation of the mitochondria plays a key role in wear and tear, slowing down and aging of the brain and body.

Alpha lipoic acid R+ regenerates other antioxidants such as vitamin C, vitamin E, and glutathione, the No. 1 intracellular detoxifier. It represents the most important limiting factor in making the most important fuel for mitochondria: Acetyl Co enzyme A. Oxidation of the mitochondria plays a key role in wear and tear, slowing down and aging of the brain and body.

Properties of R+ alpha lipoic acid

Alpha lipoic acid R+ has the particularity of :

• protect and optimize the functioning of the mitochondria

• protect and revitalize neural and physical capacities at a higher level.

R+ Alpha Lipoic Acid protects and strengthens in particular :

• brain functions

• disturbances in blood sugar levels

• liver cell function

• the cells of the eyes, heart, kidneys

• blood vessels

• the cells of the organism from the toxic action of heavy metals

Increase in the metabolic efficiency of cells

R+ alpha lipoic acid completely restores the decrease in oxygen consumption due to age. It increases the membrane potential of the mitochondria, allowing each cell to maintain optimal voltage, function and interaction with other cells.

• R+ alpha-lipoic acid increases the metabolic activity (ambulatory activity) of the cells by a factor of 3 compared to a control group.

• Fat oxidation, measured by the level of malondialdehyde (cellular MDA-toxic), is maltilated by 5 in the cells of rats not taking R+ alpha-lipoic acid.

• Taking R+ alpha lipic acid reduces MDA levels very significantly.

• The levels of glutathione (a powerful antioxidant) and vitamin C are significantly lowered in the liver cells of older organisms. The intake of R+ alpha lipic acid regulates this decrease as well as glutathione and vitamin C levels. (FASEB J. 1999)

Detoxification

R+ alpha-lipoic acid stimulates the production of glutathione, the number one detoxifier of our cells. Each cell of our organism is an autonomous entity in which there is a zone in charge of producing energy but also a zone dedicated to the detoxification of the cell's waste. Glutathione prevents cell intoxication and fouling of the body.

R+ alpha-lipoic acid allows a better bioavailability of cysteine and a greater incorporation of this amino acid inside the cells for an accelerated synthesis of glutathione.

> Alpha Lipoic Acid R + Acetyl Cysteine = Glutathione

Blood sugar control

Alpha-lipoic R+ acid regulates disturbances in blood sugar levels. It's an insulin mimetic. R+ Alpha Lipoic Acid allows the ingested sugars to be quickly assimilated by the cells, not to stagnate in the bloodstream, avoiding damage to the cells and causing a drop in energy.

Two scientific studies carried out in 1995 show that alpha lipoic acid increases insulin sensitivity by 27% to 51% in 10 days.

> R+ Alpha Lipoic Acid stimulates acetylcholine, the neurotransmitter of memory.

Heavy metal chelation

R+ alpha-lipoic acid is the most suitable nutrient for capturing, capturing and removing heavy metals. Its "pincer" molecular structure allows it to capture heavy metals. The action of alpha-lipoic acid is not limited to "moving" heavy metals from one cell compartment to another as other products do.

R+ alpha-lipoic acid truly sequesters heavy metals until they are eliminated from the body.

It easily crosses the blood-brain barrier unlike other chelators in order to dislodge heavy metals located in the intracellular.

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Neurochem Res. Long. J, Gao F, 2009

Diabetologica et Drug Research.